Our intestinal microbes do everything. They rush to digest. It constitutes our mood. They train our immune devices. It even replaces the cells-or at least, its secondary products, where recent research reveals that the microbial interlocuties that are left behind from the digestion of the amino acids can restore the cells that produce hormones in our courage.
I mentioned in International Journal of Molecular Sciencesand The results showed that these hormone -producing cells are reduced in individuals with obesity, and may contribute to obesity metabolic conditions. But the research also indicates that these cuts can be reversed thanks to the intestine microbiome, which creates the possibility of treatments or future treatments for metabolic conditions (and for potential drugs such as OzemPIC).
“The results we have reached indicate that the microbial microblogs derived from food tribly can reflect obesity discounts in the intestinal cells that release hormones at the Marshall University in West Virginia, in A, A, in A. press release. “This indicates a possible treatment strategy that enhances the intestinal microbes to improve the results of obesity.”
Read more: Here is how OzemPIC actually works for weight loss
The power of microbial fluctuations
Terptophan – The food amino acid in Turkey (and the least famous in poultry and other meat, as well as in dairy, eggs, nuts and seeds) – helps your body produce hormones such as melatonin and serotonin, which affect your sleep, your mood, and your witness.
But the new study indicates that tryptophan can play a more important role in your metabolism more than previously believed, as its microbial receptors (mainly, its secondary products after breaking them by the intestine microbium) can be used to restore hormone -producing cells in the intestine called the intended falcon cells.
Since these cells usually produce the bibli -1 hormones similar to glucagon, or GLP -1, which control the production and desire of insulin, the lack of these cells can contribute to insulin resistance, increased appetite, and poor metabolism.
Test of the disciplophan fluctuations
Portachor said in the statement that the authors of the new study have turned into a group of mice models and human intestinal organs, or “small channels”, which were presented to imitate “architecture and synthetic complexity of the original human intestine.”
When stimulating obesity in the mice and then estimated EECS, the researchers found that obesity is linked to a decrease of approximately 60 percent in EECS in the intestine. Meanwhile, by dealing with miniature fuel using tryptophan receptors and then by estimating their EECS, they found that the secondary products of the amino acids are linked to an increase of about 100 percent in EECS, which means that these signs can recover the cells produced by hormones in the intestine, and thus produce them from the GLP-1.
Read more: Other GLP-1 effects may outperform the benefits
An alternative to OzemPIC?
Combating, the results showed that tryptophan receptors are a promising way to improve metabolism health in individuals with obesity and obese metabolic conditions, although future tests and clinical trials will definitely need to confirm their beneficial effects.
Of course, GLP-1 hormones may look familiar, as they are the GLP-1 alarm model like OzemPic. By simulating the natural hormone, these artificial hormones control insulin and appetite and enhance weight loss.
Therefore, any treatments or treatments derived from tryptophan receptors can be an alternative to other GLP-1 receptors in the future that can improve metabolism healthy naturally, not by creating the GLP-1 artificial hormone, but by creating more cells that produce GLP-1 in the first place.
This article does not provide medical advice and should be used for media purposes only.
Read more: A new molecule competes with OzemPIC, which indicates less side effects
Article sources
Our book is in DiscoverMagazine.com Use studies reviewed by peers and high -quality sources of our articles, and review our editors for scientific accuracy and editorial standards. Review the sources used below for this article:
Sam Walters is a journalist covering archeology, excavation science, environment and development to discover, along with a variety of other topics. Before joining the Discover team as an assistant editor in 2022, Sam studied the press at Northwestern University in Ivston, Illinois.